Search results for " Complex Partial"

showing 5 items of 5 documents

Intracranial measurement of current densities induced by transcranial magnetic stimulation in the human brain

2003

Transcranial magnetic stimulation (TMS) is a non-invasive technique that uses the principle of electromagnetic induction to generate currents in the brain via pulsed magnetic fields. The magnitude of such induced currents is unknown. In this study we measured the TMS induced current densities in a patient with implanted depth electrodes for epilepsy monitoring. A maximum current density of 12 microA/cm2 was recorded at a depth of 1 cm from scalp surface with the optimum stimulation orientation used in the experiment and an intensity of 7% of the maximal stimulator output. During TMS we recorded relative current variations under different stimulating coil orientations and at different points…

Electromagnetic fieldAdultMaterials sciencemedicine.medical_treatmentModels NeurologicalNuclear magnetic resonanceElectromagnetic FieldsEpilepsy Complex PartialmedicineHumansGeneral NeuroscienceSkullBrainTranscranial Magnetic StimulationElectric StimulationIntensity (physics)Magnetic fieldElectromagnetic inductionElectrodes ImplantedTranscranial magnetic stimulationElectromagnetic coiltranscranial magnetic stimulatione depth electrodes epilepsyBrain InjuriesFemaleCurrent (fluid)ArtifactsCurrent densityNeuroscience
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In the rat maximal dentate activation model of partial complex epilepsy, the anticonvulsant activity of levetiracetam is modulated by nitric oxide-ac…

2009

The effects of nitric oxide-active drugs on the anticonvulsant action of the antiepileptic drug levetiracetam in an experimental model of partial complex seizures named maximal dentate gyrus activation were studied in rats. Levetiracetam was given alone or in combination with 7-nitroindazole, a preferential inhibitor of neuronal nitric oxide synthase, or with L: -arginine, the precursor of nitric oxide synthesis. The maximal dentate activation parameters were the time of latency and the durations of maximal dentate activation and afterdischarge responses. The administration of levetiracetam showed an anticonvulsant effect that was increased when given in combination with 7-nitroindazole. Th…

Male7-NitroindazoleIndazolesLevetiracetamMaximal dentate activation - Nitric oxide - Levetiracetam - Modulation - 7-Nitroindazolemedicine.medical_treatmentNitric Oxide Synthase Type IPharmacologyArginineNitric OxideSettore BIO/09 - FisiologiaNitric oxideEpilepsychemistry.chemical_compoundEpilepsy Complex PartialmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats WistarMaximal dentate activation Nitric oxide Levetiracetam Modulation 7-NitroindazoleBiological PsychiatryDose-Response Relationship DrugChemistryDentate gyrusPiracetammedicine.diseaseEffective dose (pharmacology)PiracetamRatsPsychiatry and Mental healthDisease Models AnimalDrug CombinationsAnticonvulsantNeurologyDentate GyrusAnticonvulsantsNeurology (clinical)Levetiracetammedicine.drugJournal of neural transmission (Vienna, Austria : 1996)
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Modulatory effects of nitric oxide-active drugs on the anticonvulsant activity of lamotrigine in an experimental model of partial complex epilepsy in…

2007

Abstract Background The effects induced by administering the anticonvulsant lamotrigine, the preferential inhibitor of neuronal nitric oxide synthase 7-nitroindazole and the precursor of NO synthesis L-arginine, alone or in combination, on an experimental model of partial complex seizures (maximal dentate gyrus activation) were studied in urethane anaesthetized rats. The epileptic activity of the dentate gyrus was obtained through the repetitive stimulation of the angular bundle and maximal dentate gyrus activation latency, duration and post-stimulus afterdischarge duration were evaluated. Results Either Lamotrigine (10 mg kg-1) or 7-nitroindazole (75 mg kg-1) i.p. administration had an ant…

MalePARTIAL COMPLEX EPILEPSYIndazolesArgininemedicine.medical_treatmentLamotriginePharmacologyArginineLamotrigineNitric OxideSettore BIO/09 - Fisiologialcsh:RC321-571Nitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundEpilepsy Complex PartialmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats Wistarlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNitric oxide Lamotrigine epilepsy controlbiologyTriazinesExperimental modelGeneral NeuroscienceDentate gyruslcsh:QP351-495BrainElectric StimulationRatsNitric oxide synthaseDisease Models Animallcsh:Neurophysiology and neuropsychologyAnticonvulsantnervous systemchemistryDentate Gyrusbiology.proteinAnticonvulsantsNitric Oxide SynthaseResearch Articlemedicine.drugBMC Neuroscience
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Cholecystokinin-8 sulfate modulates the anticonvulsant efficacy of vigabatrin in an experimental model of partial complex epilepsy in the rat.

2009

Summary Purpose:  We evaluated the possible additive effect induced by the administration of the anticonvulsant vigabatrin (VGB) and cholecystokinin-8 sulfate (CCK-8S) on an experimental model of partial complex seizures (maximal dentate gyrus activation, MDA). Moreover, the functional involvement of γ-aminobutyric acid (GABA) neurotransmission was tested by iontophoretically administering bicuculline (GABA receptor antagonist) in the dentate gyrus. Methods:  Urethane anesthetized rats were pretreated with VGB (50, 100 or 200 mg/kg, i.p.) or CCK-8S (8 nmol/kg, i.p.) alone or coadministered with VGB (50 mg/kg, i.p.). Dentate gyrus epileptic activity was obtained through the repetitive electr…

Malemedicine.medical_treatmentStimulationConvulsantsNeurotransmissionPharmacologyBicucullineRat Partial epilepsy Vigabatrin Cholecystokinin-8 sulfate ControlVigabatrinDrug Administration ScheduleSincalideVigabatrinEpilepsy Complex PartialmedicineReaction TimeAnimalsRats WistarEvoked PotentialsNootropic AgentsAnalysis of VarianceIontophoresisDose-Response Relationship DrugChemistryDentate gyrusDrug SynergismBicucullineGABA receptor antagonistElectric StimulationRatsDisease Models AnimalAnticonvulsantNeurologyAnesthesiaDentate GyrusAnticonvulsantsDrug Therapy CombinationNeurology (clinical)medicine.drugEpilepsia
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Hyperammonemic encephalopathy after urinary diversion. Diet therapy

2016

medicine.medical_specialtyDiet therapymedicine.medical_treatment030232 urology & nephrologyUrinary DiversionPlant Proteins DietaryGastroenterologyIntestinal absorption03 medical and health sciencesEpilepsy Complex Partial0302 clinical medicineAmmoniaInternal medicinemedicineHumansHyperammonemia030212 general & internal medicineAgedAcidosisCarcinoma Transitional CellCARCINOMA TRANSITIONAL CELLBrain Diseases Metabolicbusiness.industryUrinary diversionHyperammonemiaHydrogen-Ion Concentrationmedicine.diseaseSurgeryIntestinal AbsorptionUrinary Bladder NeoplasmsFemaleDietary Proteinsmedicine.symptomAcidosisHyperammonemic encephalopathybusinessEndocrinología y Nutrición (English Edition)
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